Eagle syndrome (ES) is a rare and diagnostically challenging condition caused by pathological elongation of the styloid process or ossification of the styloidhyoid ligament. Frequently presenting with unilateral orofacial/neck pain triggered by neck extension, an atypical presentation is syncopal events due to impingement and carotid sinus stimulation. Rapid identification and surgical management is pivotal to mitigate significant morbidity and mortality from such cardiac events. We report an interesting case of recorded asystole resulting from ES in an otherwise well patient. We present a case of a 47-year-old who experienced multiple asysolic events from baroreceptor compression in the setting of new diagnosis of ES. The patient presented with recurrent severe unilateral neck pain when eating, followed by a witnessed syncope. He had no history of pharyngeal trauma, tonsillectomy or cardiac risk factors. Whilst in ED, he had a further witnessed non-responsive episode preceded by diaphoresis and pallor during which 20 seconds of asystolic arrest was captured on telemetry. CT Angiogram demonstrated no signs of carotid dissection or compression, but severe calcification of the left stylohyoid ligament from the styloid process to hyoid bone and a 7cm styloid process. In discussion with Cardiology, a pacemaker device was not felt to be indicated pre-operatively. He therefore underwent an uncomplicated elective left transoral styloidectomy which confirmed both styloid elongation and a grossly calcified ligament. He has experienced no further syncopal events in the acute post-operative period and no facial nerve injury. Asystole is a very rare complication of ES and case reports are scarce within the literature. This case highlights the need for all clinicians to be cognisant of ES as a differential diagnosis in unexplained syncope. Rapid recognition of symptoms and signs of ES and prompt escalation to styloidectomy can prevent significant morbidities such as trauma, end organ ischaemia, and even sudden cardiac death.